HEPATITIS B INFECTIOUS DISEASE CAUSED HEPATITIS B VIRUS LIVER CAUSE ACUTE CHRONIC INFECTION PEOPLE SYMPTOMS INITIAL DEVELOP RAPID ONSET VOMITING YELLOWISH SKIN TIREDNESS DARK URINE ABDOMINAL PAIN LAST WEEKS INFECTION IN DEATH DAYS INFECTED BIRTH CHRONIC LESS THAN THE AGE CHRONIC DISEASE CIRRHOSIS LIVER CANCER EVENTUALLY COMPLICATIONS DEATH THE VIRUS EXPOSURE BLOOD BODY FLUIDS CONTACT PEOPLES CHILDHOOD METHOD ACQUIRED AREAS THE DISEASE COMMON INTRAVENOUS DRUG USE SEXUAL INTERCOURSE ROUTES OF INFECTION RISK FACTORS BLOOD TRANSFUSIONS DIALYSIS LIVING TRAVEL THE INFECTION RATE TATTOO ACUPUNCTURE NUMBER CASES LESS STERILITY VIRUSES CANNOT SPREAD HOLDING HANDS SHARING EATING DIAGNOSIS TESTING THE BLOOD PARTS ANTIBODIES HEPATITIS VIRUSES C THE WORLD HEALTH ORGANIZATION FIRST DAY FULL VACCINE GAVE THE VACCINE NATIONAL AS OF TESTED TRANSFUSION CONDOMS BE USED TO PREVENT CARE PERSON ANTIVIRAL MEDICATION TENOFOVIR INTERFERON USEFUL DRUGS LIVER TRANSPLANTATION SOMETIMES THE WORLD POPULATION ONE POINT THEIR LIVES INFECTIONS EAST ASIA AFRICA ADULTS RATES EUROPE NORTH AMERICA SERUM HEPATITIS RESEARCH LOOKING CREATE FOODS HBV VACCINE AFFECT GREAT APES ACUTE INFECTION VIRUS VIRAL HEPATITIS ILLNESS BEGINS GENERAL LOSS OF APPETITE BODY MILD DEVELOPMENT OF JAUNDICE INDICATION POSSIBLE SYMPTOM HEPATITIS VIRUS LASTS IMPROVES LIVER DISEASE FULMINANT HEPATIC FAILURE ASYMPTOMATIC CHRONIC INFLAMMATION LEADING PERIOD TYPE DRAMATICALLY HEPATOCELLULAR CARCINOMA ALCOHOL AS IT RISK MEMBRANOUS GLOMERULONEPHRITIS OUTSIDE PRESENT SYNDROME ACUTE NECROTIZING VASCULITIS POLYARTERITIS NODOSA PAPULAR ACRODERMATITIS SETTING THE ONSET FEATURES RASH POLYARTERITIS PERSIST DURATION NEPHROPATHY HEMATOLOGICAL DISORDERS ESSENTIAL MIXED CRYOGLOBULINEMIA APLASTIC ANEMIA TRANSMISSION RESULTS TIMES SEXUAL BLOOD TRANSFUSION HUMAN BLOOD CONTAMINATED NEEDLES VERTICAL TRANSMISSION MOTHER CHILD POSITIVE HBSAG PASSING OFFSPRING HIGH AS IF THE MOTHER HBEAG FAMILY MEMBERS MUCOUS MEMBRANE SECRETIONS SALIVA REPORTED IDENTIFIABLE PROPER APPEAR TO MOTHER DETECTED CHRONIC HEPATITIS B INCUBATION PERIOD MEMBER HEPADNAVIRUS FAMILY PARTICLE VIRION OUTER LIPID ENVELOPE ICOSAHEDRAL NUCLEOCAPSID CORE PROTEIN VIRAL DNA DNA POLYMERASE REVERSE TRANSCRIPTASE EMBEDDED PROTEINS INVOLVED BINDING ENTRY SUSCEPTIBLE ANIMAL VIRUSES INFECTING CELLS HEPATOCYTE DANE PARTICLES ADDITION SPHERICAL BODIES SERUM THE SURFACE ANTIGENS IN EXCESS THE LIFE CYCLE GENOME THE DNA FULLY FULL LENGTH STRAND NUCLEOTIDES SHORT COMPLEMENTARY MRNA NUCLEUS SOON THE CELL RENDERED COMPLETION SENSE STRAND PROTEIN MOLECULE SEQUENCE RNA BASES ENDS REJOINED GENES ENCODED THE CORE GENE START CODON UPSTREAM PROCESSING RARE STRAINS HEPATITIS B VIRUS PRECORE MUTANT POLYMERASE CODES ANTIGEN OPEN READING FRAME IN FRAME START CODONS DIVIDE MULTIPLE START CODONS POLYPEPTIDE ORDER SURFACE INSIDE FUNCTION GENE X UNDERSTOOD PROMOTE CELL GROWTH PARARETROVIRUS RETROVIRUS REVERSE TRANSCRIPTION REPLICATION NTCP HOST GENOMIC DNA HAS TO DOUBLE STRANDED COVALENTLY CLOSED CCCDNA TEMPLATE TRANSCRIPTION OF LARGEST LONGER USED TO CAPSID CORE PROTEIN UNDERGO GO ON PROGENY PRODUCE TRANSPORTED CYTOPLASM SYNTHESIZES DIVIDED MAJOR SEROTYPE ANTIGENIC EPITOPE PRESENTED ENVELOPE PROTEINS EIGHT OVERALL NUCLEOTIDE SEQUENCE VARIATION DISTINCT GEOGRAPHICAL TRACING THE EVOLUTION DIFFERENCES SEVERITY COURSE LIKELIHOOD RESPONSE FUNCTIONS HEPATOCYTES FUNCTIONAL RECEPTOR EVIDENCE RELATED DUCK HEPATITIS B VIRUS CARBOXYPEPTIDASE D BIND THE HOST CELL DOMAIN SUBSEQUENTLY INTERNALIZED ENDOCYTOSIS RECEPTORS CELLULAR RECEPTORS EXIST IMMUNE CAUSES DAMAGE INNATE IMMUNE RESPONSE PLAY ROLE ADAPTIVE PARTICULAR CYTOTOXIC T LYMPHOCYTES LIVER INJURY CTLS BY KILLING ANTIVIRAL CYTOKINE PURGE LIVER DAMAGE INITIATED MEDIATED PLATELETS THE SITE FACILITATE ACCUMULATION ASSAY DETECTION VIRUS INFECTION INVOLVE BLOOD TEST DETECT INTERPRETATION OF ASSAYS SCREEN PRESENCE INNER CORE ENCLOSING CORE PARTICLE CORE ANTIGEN HBCAG THIS WINDOW REMAINS CLEARING IGM ANTIBODIES THE ONLY DIAGNOSTIC TOTAL APPEARANCE TRADITIONALLY HOSTS HIGHER VIRAL REPLICATION INFECTIVITY DO NOT RULE ALWAYS HOLD THE NATURAL ARISE IMMEDIATELY AFTERWARDS CONVERSION DECLINE ABLE CLEAR IGG ANTIBODIES ANTI HBC THE WINDOW NEGATIVE VACCINATED PREVIOUSLY INDIVIDUALS MONTHS ELEVATED ALANINE AMINOTRANSFERASE INFLAMMATION THE IMMUNE CLEARANCE PHASE SEROCONVERTED LITTLE MULTIPLICATION HENCE TRANSMITTING DEVELOPED MEASURE AMOUNT VIRAL LOAD PERSONS STATUS MONITOR GROUND GLASS HEPATOCYTE PREVENTION INFANTS THE UNITED DEFINED ANTIBODY CONCENTRATION EFFECTIVE CHILDREN PERCENT DROPS THE PROTECTION FALL BELOW FOR ALL COMBINATION HEPATITIS B IMMUNE GLOBULIN SHOULD VERIFY IMMUNIZED ASSISTED REPRODUCTIVE TECHNOLOGY SPERM WASHING NECESSARY MALES UNLESS FEMALE PARTNER EFFECTIVELY FEMALES HEPATITIS SPONTANEOUS CONCEPTION HIGH RISK SCREENING INTRAVENOUS DRUG USERS MEN WHO HAVE SEX WITH MEN SOMEONE NORMAL IMMUNE SYSTEM DOCUMENTED FEWER WHOSE TAKES AGGRESSIVE IMMUNOCOMPROMISED ON THE OTHER HAND REDUCE MARKER CANDIDATES MEDICATION NONE AV