ALOPECIA AREATA PRACTICE PATHOPHYSIOLOGY PERSPECTIVE DRUGS DISEASES EDUCATION ACADEMY ENGLISH DEUTSCH FRANAIS PORTUGUS LOG IN SIGN REGISTER RESULTS CLOSE PLEASE CONFIRM LOG OUT MEDSCAPE IF YOU YOU WILL ENTER YOUR USERNAME PASSWORD THE NEXT TIME CANCEL DERMATOLOGY CHIEF EDITOR DIRK SHARE EMAIL PRINT FEEDBACK FACEBOOK TWITTER LINKEDIN OVERVIEW ESSENTIALS BACKGROUND EPIDEMIOLOGY PROGNOSIS PATIENT EDUCATION SHOW PRESENTATION PHYSICAL CAUSES HISTOLOGIC FINDINGS APPROACH TOPICAL TREATMENTS SYSTEMIC MODALITIES SURGICAL CARE PREVENTION MEDICATION SUMMARY IMMUNOMODULATORS GLUCOCORTICOIDS VASODILATORS MEDIA GALLERY REFERENCES RECURRENT TYPE HAIR LOSS AFFECT AREA MANIFEST BENIGN CONDITION PATIENTS CAUSE EMOTIONAL THE IMAGES VIEW OPHIASIS PATTERN SIGNS AND SYMPTOMS EXPERIENCE BURNING SENSATION PRURITUS THE CONDITION FIRST APPEARS PATCH PATCHES MULTIPLE CORRELATION NUMBER ONSET SEVERITY FREQUENCY PARTICULAR SCALP BEARD MALES EYEBROWS EXTREMITIES CONDITIONS ATOPIC DERMATITIS VITILIGO THYROID DISEASE DOWN SYNDROME PSYCHIATRIC DISORDERS PERSONALITY DISORDERS PARANOID DISORDERS EVENTS THE 6 MONTHS CLASSIFIED RETICULAR COALESCE LOWER BACK THE SCALP SPELLED THE HEAD ALOPECIA TOTALIS ALOPECIA UNIVERSALIS COMPLETE LOSS OF HAIR AREAS NAIL REPORTED ABNORMALITIES TRACHYONYCHIA ONYCHORRHEXIS ONYCHOMADESIS LEUKONYCHIA DIAGNOSIS BIOPSY HELPFUL CLINICAL DIAGNOSIS LESS MANAGEMENT RECURRENCES TOPICAL CORTICOSTEROIDS CORTICOSTEROID THERAPY LESS THAN ADMINISTRATION SYRINGE NEEDLE TRIAMCINOLONE ACETONIDE CONCENTRATIONS CONCENTRATION THE FACE OF 5 SUFFICIENT SPREAD COVER INJECTION WEEKS CHILDREN CANNOT TOLERATE FLUOCINOLONE ACETONIDE CREAM TWICE DAILY BETAMETHASONE DIPROPIONATE REFRACTORY CLOBETASOL PROPIONATE OCCLUSION PLASTIC FILM HELPED MINORITY MUST MINIMUM OF 3 MAINTENANCE THERAPY NECESSARY PREDNISONE AGENT CHOICE ADVERSE EFFECTS INITIAL DOSE SO HIGH RELAPSE DISCONTINUATION IMMUNOTHERAPY DEFINED INDUCTION ALLERGIC CONTACT DERMATITIS TOPICAL APPLICATION POTENT CONTACT ALLERGENS AGENTS SQUARIC ACID DIPHENCYPRONE ANTHRALIN OVERNIGHT MINOXIDIL APPEARS EFFECTIVE THE TREATMENT DISEASE LITTLE BENEFIT SOLUTION BE MORE NO MORE DROPS EXTENT SEEN APPLICATION PSORALEN PLUS PUVA THERAPIES CASES CYCLOSPORINE LIMITED EFFICACY TACROLIMUS METHOTREXATE MIXED COSMETIC TREATMENT USED TO CAMOUFLAGE SESSIONS HOUR FOR EACH PATIENT USEFUL TREMENDOUS DISTRESS PREVIOUS EXACT PATHOPHYSIOLOGY OF REMAINS ACCEPTED HYPOTHESIS AUTOIMMUNE GENETICALLY AUTOIMMUNITY EVIDENCE THE PROCESS ANTIBODIES HAIR FOLLICLE STRUCTURES CONTROL IMMUNOFLUORESCENCE HAIR FOLLICLES IN AS AS 90 AUTOANTIBODY RESPONSE TARGETS OUTER ROOT SHEATH THE STRUCTURE THE HAIR WHETHER PLAY DIRECT ROLE PATHOGENESIS EPIPHENOMENON LYMPHOCYTIC CELLS LYMPHOCYTES DEPLETION SUBTYPES HAIR DUNDEE EXPERIMENTAL BALD MODEL THE ANIMALS SUBSEQUENTLY ONCE POPULATION REPLETE FACT ANIMALS SUGGESTS INVOLVED TOTAL NUMBERS NORMAL HUMANS REINFORCE ONTO SCID COMBINED IMMUNODEFICIENCY MICE DEVOID IMMUNE CELL AUTOLOGOUS CULTURED GRAFTS LOSS FOLLICLE TRIGGER HAIR SIMILAR EXPERIMENT NUDE FAILED SERUM INTRAVENOUSLY STUDY DEPOSITION IMMUNOGLOBULIN COMPLEMENT GRAFTED SKIN RESEARCH TRANSFER ALOPECIA THYROID DISEASES RETROSPECTIVE REVIEW PRESENTED ACADEMIC MEDICAL CENTERS BOSTON DIAGNOSES DIABETES MELLITUS INFLAMMATORY BOWEL DISEASE SYSTEMIC LUPUS ERYTHEMATOSUS RHEUMATOID ARTHRITIS PSORIASIS PSORIATIC ARTHRITIS ATOPY CONTACT DERMATITIS ECZEMA MENTAL HEALTH PROBLEMS HYPERLIPIDEMIA HYPERTENSION GERD CONCLUSION BENEFICIAL SUBTYPE HAIR GROWTH DETECTION AUTOANTIBODIES ABILITY GRAFTING IMMUNOSUPPRESSED CERTAIN FACTORS TRIGGER AUTOIMMUNE REACTION TARGET ADDING IMMUNOLOGIC THE OUTCOME GENETICS GENETIC PREDISPOSITION THE FREQUENCY POSITIVE FAMILY HISTORY IN CONTROL HIGHER REPORTS TWINS DEGREE GENES AMOUNT FOCUSED HUMAN LEUKOCYTE ANTIGEN MARKER GENERAL PRESENT GENE INTEREST THE INTERLEUKIN 1 RECEPTOR ANTAGONIST THE HIGH ASSOCIATION OF CHROMOSOME GENETIC DETERMINING POLYGENIC RATHER SINGLE GENE THE ROLE ENVIRONMENTAL FACTORS TRIGGERING TO BE DETERMINED CYTOKINES INTERLEUKIN 1 TUMOR NECROSIS FACTOR INHIBITORS MICROSCOPIC EXAMINATION CHANGES INNERVATION THE MODIFICATION REPORT ITCHING PAIN THE POSSIBILITY ALTERATIONS IN THE PERIPHERAL NERVOUS NEUROPEPTIDE CALCITONIN PEPTIDE CGRP THE IMMUNE SYSTEM CHEMOTAXIS INHIBITION LANGERHANS CELL ANTIGEN PRESENTATION PROLIFERATION VASODILATATION ENDOTHELIAL CUTANEOUS SUBSTANCE P VASOACTIVE INTESTINAL POLYPEPTIDE THE STUDY BASAL BLOOD FLOW GREATER INTRADERMAL SHED LIGHT ON THE SIGNIFICANCE OF VIRAL ETIOLOGY HYPOTHESES EXPLAIN SUPPORT BELIEVED INFECTIOUS MICROBIAL ISOLATE PREVALENCE THE GENERAL LIFETIME RISK DERMATOLOGISTS RACE RACES INCREASE ETHNIC SEX DATA SEX RATIO RATIO SMALLER FEMALE BIRTH DECADES CONGENITAL PEAK PEOPLE YOUNGER OLDER 40 YEARS PERSONAL APPEARANCE OPENLY ADDRESSING HELPING COPE THE NATURAL HISTORY OF FOCAL ESTIMATES ADVERSE PROGNOSTIC YOUNG AGE FACTOR INFORM THE CHRONIC RELAPSING NATURE FIND EXPLANATION THIS IS HAPPENING DONE NOTHING WRONG SHOULD SUPPORT GROUPS AVAILABLE THE 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